healing, immune function, fecundity and others. The focus, here will be on forcing a default state to mitochondrial regenesis, which is the heart and soul of life extension and the inhibition of cancer cell induction and maintenance. Thus, the discussion will completely avoid the well worn schoolmarm admonitions such as ‘eat your vegetables,’ ‘take your
vitamins,’ ‘exercise regularly,’ ‘drink plenty of water,’ ‘keep your weight down,’ ‘don’t eat between meals,’ ‘brush your teeth after every meal,’ ‘don’t eat anything that you can’t fit into your mouth,’ ‘don’t ingest it if you can’t pronounce i,’ ‘holy cow, that sure is a big fish’ and other common sense standard fare that we won’t even mention, here.
It is difficult to tell how much of dietary resveratrol is neogenic and how much is regenic. It cannot be heavily neogenic due to its TOR bypass, catabolic and anti-oxidant functions plus its average life expectancy increasing and rejuvenative outcomes in the absence of increased cancer induction or any true life length reduction. Standard dietary resveratrol also cannot be very strongly regenic because there is no appreciable up regulation in AMPK, no real reduction in cancer incidence, no increase in cancer cell apoptosis and no true life extension. However, much mitochondrial biogenesis is observed, but how much of this is neogenesis converted to regenesis is unknown because most investigators were unaware of the difference.
Progressive nutriceutical supply companies recognize this and are actively pursuing remedies. The essential problem is simple. When resveratrol is eaten, well over 90% of it is sulfonated and glucuronidated in the intestines and in the liver via the hepatic portal